The Ramachandran Lab
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For a full list of publications click here
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​Molecular basis for activation and biased signalling at the thrombin-activated GPCR Proteinase Activated Receptor-4 (PAR4)
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Targeting a PAR4 carboxyl terminal motif to regulate platelet function.
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GPCR mediated EGFR transactivation regulates TRPV4 action in the vasculature. 
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Neutrophil elastase and proteinase-3 trigger G protein-biased signaling through proteinase-activated receptor-1 (PAR1).
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TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
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Neutrophil elastase acts as a biased agonist for proteinase-activated receptor-2 (PAR2).
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Reviews

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Proteinases, their extracellular targets, and inflammatory signalling. 
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Targeting proteinase-activated receptors: therapeutic potential and challenges
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Structure, function and pathophysiology of proteinase activated receptors.
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